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Published on: 8/18/2026

Can Adults Get Rickets?

Yes, adults can develop the same underlying bone-softening problem, but in adults it is called osteomalacia because rickets specifically affects the growth plates in children's growing bones. It is usually caused by low vitamin D, calcium, or phosphate from poor sun exposure, diet, malabsorption conditions, kidney or liver disease, certain medications, or inherited disorders, and symptoms often include deep bone pain, muscle weakness, waddling gait, and fractures from minor injury. Adults who had untreated rickets in childhood may also carry lasting bowed legs, spinal curvature, or dental problems. Because these symptoms overlap with arthritis, fibromyalgia, and other conditions, there are several important factors and warning signs to consider before assuming a cause. See below to understand more.

If bone pain, weakness, or unexplained fractures are affecting you, the fastest way to start sorting out what is going on is a free, instant, online symptom check that reviews your symptoms in minutes and points you toward the right next step, whether that is a vitamin D test, a primary care visit, or urgent evaluation. It takes just a few minutes, costs nothing, and can help you walk into your appointment with clearer questions and less guesswork.

Last reviewed for medical accuracy: 08/18/2026

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Explanation

Can Adults Get Rickets?

Rickets is often described as a childhood bone disorder, but you may be wondering, “can adults get rickets?” While true rickets—characterized by soft, weakened growth plates—is specific to children and adolescents, adults can develop a similar condition called osteomalacia. Both stem primarily from inadequate vitamin D, calcium or phosphate, leading to softening of the bones. Understanding risk factors, signs, prevention and treatment can help you protect your bone health at any age.


Why Rickets Is Rare in Adults

  • In children, rickets affects the growth plates (epiphyses) of long bones, causing deformities like bowed legs.
  • Once growth plates close after puberty, true rickets can no longer develop.
  • Adults with vitamin D or mineral deficiencies develop osteomalacia—a softening of already mature bone rather than changes in bone shape.
  • Despite different names, the underlying problem (faulty bone mineralization) is the same.

Risk Factors for Adult Bone-Softening Disorders

Even though “can adults get rickets” in the strict sense is uncommon, osteomalacia shares many causes and can lead to similar symptoms. Key risk factors include:

  • Vitamin D deficiency

    • Limited sun exposure (indoor lifestyle, high-latitude living, use of sunscreens)
    • Darker skin pigmentation (melanin reduces vitamin D synthesis)
    • Strict vegan diets without fortified foods
  • Calcium or phosphate deficiency

    • Malabsorption syndromes (celiac disease, inflammatory bowel disease)
    • Bariatric surgery reducing nutrient uptake
  • Kidney or liver disease

    • Impaired conversion of vitamin D to its active form
    • Loss of phosphate through damaged kidneys
  • Certain medications

    • Anticonvulsants (phenytoin, phenobarbital)
    • Some HIV treatments
    • Long-term glucocorticoids (“steroid” drugs)
  • Genetic or rare metabolic disorders

    • Hypophosphatasia (low alkaline phosphatase activity)
    • Inherited vitamin D–resistant conditions

Signs and Symptoms to Watch For

Osteomalacia can be subtle at first, but recognizing early clues helps you seek treatment before serious complications arise.

Early symptoms may include:

  • Dull, aching bone pain—often in the lower back, pelvis or legs
  • Muscle weakness, especially in the thighs and shoulders
  • Difficulty walking or frequent falls

As the condition progresses:

  • Bone tenderness when pressure is applied
  • Increased risk of stress fractures in hips, ribs and wrists
  • Dental problems (soft enamel, cavities)

If you’re asking “can adults get rickets” because you’ve noticed persistent bone pain or weakness, consider a free, online symptom check, using the doctor approved Ubie Symptom Checker.


How Osteomalacia Is Diagnosed

Diagnosis combines your clinical history, physical exam and laboratory tests:

  1. Medical history & physical exam

    • Dietary habits, sun exposure, absorption issues, medication use
    • Assessment of muscle strength and gait
  2. Blood tests

    • 25-hydroxyvitamin D (best measure of vitamin D status)
    • Calcium, phosphate and alkaline phosphatase levels
    • Parathyroid hormone (PTH) to evaluate secondary hyperparathyroidism
  3. Imaging studies

    • X-rays may show Looser’s zones (pseudofractures)
    • Dual-energy X-ray absorptiometry (DEXA) for bone density
  4. Additional tests (if needed)

    • Kidney and liver function tests
    • Genetic or metabolic panels for rare causes

Treatment Strategies

Treating osteomalacia focuses on reversing deficiencies and addressing underlying causes:

  1. Vitamin D supplementation

    • High-dose oral vitamin D₂ (ergocalciferol) or D₃ (cholecalciferol)
    • Typical courses last 8–12 weeks, then maintenance doses
  2. Calcium and phosphate replacement

    • Oral calcium carbonate or citrate
    • Phosphate supplements if levels remain low
  3. Sunlight exposure

    • 10–30 minutes of midday sun, 2–3 times per week, arms and legs exposed
    • Adjust time based on skin tone, season and latitude
  4. Treat underlying conditions

    • Manage malabsorption (gluten-free diet, pancreatic enzyme replacement)
    • Review medications with your doctor; adjust or switch therapies if possible
    • Address liver or kidney disease under specialist care
  5. Physical therapy

    • Strengthening exercises to improve gait and reduce fall risk
    • Balance training and low-impact activities (walking, swimming)
  6. Monitoring

    • Repeat blood tests and imaging every 3–6 months until stable
    • Ongoing follow-up for chronic conditions

Prevention Tips

Healthy bones are built year-round. To minimize your risk:

  • Enjoy balanced, nutrient-rich meals

    • Fatty fish (salmon, mackerel), egg yolks, mushrooms
    • Fortified dairy or plant milks, cereals and orange juice
  • Optimize sun exposure safely

    • Brief, regular intervals without sunscreen, then protect skin for longer exposure
    • Aim for midday sun when UVB rays are strongest
  • Maintain a healthy body weight

    • Obesity and underweight both affect vitamin D bioavailability
  • Stay active

    • Weight-bearing exercises (walking, jogging, resistance training)
    • Improves bone density and muscle strength
  • Schedule routine checkups

    • Especially if you have risk factors like chronic digestive or kidney issues

When to Seek Professional Help

If you experience persistent bone pain, muscle weakness or signs of fractures, it’s important not to ignore them. A free, online symptom check, using the doctor approved Ubie Symptom Checker, can help you decide if you need medical attention right away.

Always consult your doctor if you notice:

  • Severe or worsening bone pain
  • Unexplained muscle weakness affecting daily activities
  • Sudden fractures or inability to bear weight
  • Symptoms of life-threatening complications (severe hypocalcemia: tingling, muscle cramps, seizures)

Key Takeaways

  • True rickets (“can adults get rickets?”) is confined to growing bones in children. Adults develop osteomalacia, with similar causes and bone-softening effects.
  • Major driver: vitamin D deficiency, but calcium, phosphate, absorption issues and chronic disease can contribute.
  • Diagnosis relies on history, exam, blood tests and imaging.
  • Treatment includes vitamin D and mineral supplementation, sun exposure, diet changes and addressing underlying conditions.
  • Prevention: balanced diet, safe sun habits, regular exercise and routine health screenings.
  • For early guidance, use a free, online symptom check, using the doctor approved Ubie Symptom Checker, then speak to a healthcare provider about any serious or persistent concerns.

If you suspect you have osteomalacia or any serious health issue, please speak to a doctor promptly. Your bones—and your overall health—are worth it.

(References)

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  • * Tebben PJ. Hypophosphatemia: A Practical Guide to Evaluation and Management. Endocr Pract. 2022 Oct;28(10):1091-1099. doi: 10.1016/j.eprac.2022.07.005. Epub 2022 Aug 6. PMID: 35940468.

  • * Ackah SA, Imel EA. Approach to Hypophosphatemic Rickets. J Clin Endocrinol Metab. 2022 Dec 17;108(1):209-220. doi: 10.1210/clinem/dgac488. PMID: 35981346; PMCID: PMC9759174.

  • * Cianferotti L. Osteomalacia Is Not a Single Disease. Int J Mol Sci. 2022 Nov 28;23(23). doi: 10.3390/ijms232314896. Epub 2022 Nov 28. PMID: 36499221; PMCID: PMC9740398.

  • * Doulgeraki A, Laurent MR. Editorial: Rickets and osteomalacia, from genes to nutrition. Front Endocrinol (Lausanne). 2023;14:1141888. doi: 10.3389/fendo.2023.1141888. Epub 2023 Jan 19. PMID: 36742385; PMCID: PMC9893633.

  • * Ito N, Hidaka N, Kato H. The pathophysiology of hypophosphatemia. Best Pract Res Clin Endocrinol Metab. 2024 Mar;38(2):101851. doi: 10.1016/j.beem.2023.101851. Epub 2023 Nov 30. PMID: 38087658.

  • * Bandgar T, Shah N. Revisiting hypophosphatemic rickets/osteomalacia. Best Pract Res Clin Endocrinol Metab. 2024 Mar;38(2):101859. doi: 10.1016/j.beem.2024.101859. Epub 2024 Jan 6. PMID: 38238129.

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